Whole-genome sequencing identifies emergence of a quinolone resistance mutation in a case of Stenotrophomonas maltophilia bacteremia.

نویسندگان

  • Theodore R Pak
  • Deena R Altman
  • Oliver Attie
  • Robert Sebra
  • Camille L Hamula
  • Martha Lewis
  • Gintaras Deikus
  • Leah C Newman
  • Gang Fang
  • Jonathan Hand
  • Gopi Patel
  • Fran Wallach
  • Eric E Schadt
  • Shirish Huprikar
  • Harm van Bakel
  • Andrew Kasarskis
  • Ali Bashir
چکیده

Whole-genome sequences for Stenotrophomonas maltophilia serial isolates from a bacteremic patient before and after development of levofloxacin resistance were assembled de novo and differed by one single-nucleotide variant in smeT, a repressor for multidrug efflux operon smeDEF. Along with sequenced isolates from five contemporaneous cases, they displayed considerable diversity compared against all published complete genomes. Whole-genome sequencing and complete assembly can conclusively identify resistance mechanisms emerging in S. maltophilia strains during clinical therapy.

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A function of SmeDEF, the major quinolone resistance determinant of Stenotrophomonas maltophilia, is the colonization of plant roots.

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عنوان ژورنال:
  • Antimicrobial agents and chemotherapy

دوره 59 11  شماره 

صفحات  -

تاریخ انتشار 2015